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Title: Optimizing ED Care for Migration-Affected Patients

Category: Administration

Keywords: Migrant, Emergency Department (PubMed Search)

Posted: 9/23/2026 by Mercedes Torres, MD (Updated: 9/23/2026)

The emergency department may be the only point of access to the health care system for migrants.  As emergency physicians, we face the challenge of balancing our ethical obligations to our patients with our responsibilities to employers, the government, and/or law enforcement. In consideration of the mandate of EMTALA and this complex role within an evolving political landscape, this publication offers four principles to guide best practices when caring for migration-affected patients in the ED. 

Click the link to review these four principles which focus on ensuring equitable care for all migration-affected patients.

Show Additional Information

Four Principles to Support Equitable ED Care for Migration-Affected Patients:

1. Practice evidence-based clinical care to reduce bias.

2. Be aware of local and federal policies and migration factors that may influence patient care trends.

3. Target screening for population-specific risks and tailor patient care services.

4. Establish community partnerships and interdisciplinary care teams.

See article for more detail with examples of each principle in action.

Show References

Stanifer K, Smith-Bellille T, Bloem C, Willis J, Aurrecoechea A.  Beyond Borders: Advancing Equitable Care in the Emergency Department for Migration-Affected Patients. The Journal of Emergency Medicine. Vol 89, 2026, Pages 109-114, https://doi.org/10.1016/j.jemermed.2026.08.005.



Title: "Everything in Excess is Opposed to Nature"

Category: Critical Care

Posted: 9/22/2026 by Caleb Chan, MD (Updated: 9/24/2026)

Question

The following is an actual case. 

An ED patient without any history of asthma or COPD is intubated to facilitate an emergent MRI. After the MRI is completed (and without abnormalities). The patient is breathing  synchronously and comfortably on minimal PS settings. Further sedation is weaned to facilitate extubation in the ED as the patient has no need for continued mechanical ventilation and no ICU requirements. All of a sudden, you are emergently called overhead to the patient's room because the patient is “blue” and hypoxic. 

These are the ventilator waveforms. 

The patient did not receive any contrast or bronchospastic medications. The ETT is clear and without obstruction.

What is going on?

Show Answer

There are a lot of issues that are apparent on this ventilator waveform. However, the focus for this pearl is the expiratory waveforms which show significant expiratory obstruction (compare to normal below). The patient is getting inspiratory volumes (VTi 553 mL in light blue bottom right corner), but limited expiratory waveforms (VTe 164 mL) . Given that the patient has no history of asthma, COPD, or reason for acute obstruction, the concern was that patient effort (as patient became more awake with decreased sedation) was actually causing expiratory obstruction. Sedation was increased and the obstruction resolved. 

The patient was subsequently diagnosed with excessive dynamic airway collapse, a condition where the posterior membrane of the tracheobronchial tree weakens and bulges anteriorly into the airways (this can sometimes be seen on expiratory CT chests). This movement is exaggerated during forced exhalation due to increased intrathoracic pressure and causes a more severe expiratory obstruction pattern compared to passive exhalation. Consequently, this can lead to a cycle of agitation, increased work of breathing, and breath-stacking/accumulation of intrinsic PEEP. 

Of note, EDAC often gets misidentified as “COPD” or “asthma,” and given its mechanism, does not respond to bronchodilators or steroids. The weakening of the posterior membrane is a chronic process and hence EDAC does not develop acutely, it simply becomes clinically apparent in an agitated intubated patient. Hence, the treatment for this patient was a sedative to calm the patient while preserving respiratory effort (i.e. dexmedetomidine) and extubating the patient to remove the exacerbating stimulus (being intubated). 

Extubating to NIPPV could be considered as a bridge granted it does not cause more agitation and increased work of breathing.

In summary, EDAC is a cause of expiratory obstruction that should  be considered in patients with evidence of obstruction and increased of work of breathing, particularly if they don’t have a history of COPD/asthma or reason for bronchospasm.



Title: A missed opportunity

Category: Cardiology

Posted: 9/20/2026 by Robert Flint, MD (Updated: 9/24/2026)

This commentary points out the missed opportunity to improve chronic hypertension care in the ED. A substantial number of patients seeking care for asymptomatic hypertension and an even larger cohort seen for other complaints who are also chronically untreated  hypertensives leave the ED without a prescription for antihypertensives. ACEP and AHA say we should be treating these people and arranging follow up.

Show References

N. E. Harrison and P. Levy, “ Chronic Uncontrolled Hypertension in the ED—We Hold This Truth to Be Self Evident,” Academic Emergency Medicine 33, no. 9 (2026): e70409, https://doi.org/10.1111/acem.70409.



Title: Perspectives and Preferences of Underrepresented in Emergency Medicine Resident Applicants: A Qualitative Study

Category: Administration

Posted: 9/19/2026 by Kevin Semelrath, MD (Updated: 9/24/2026)

This small qualitative study demonstrated that representation and hearing from other URiM residents is an important factor in recruitment during EM residency application.

Show Additional Information

M. Suh, B. Torres, A. Bhananker, et al., “ Perspectives and Preferences of Underrepresented in Emergency Medicine Resident Applicants: A Qualitative Study,” AEM Education and Training 10, no. 3 (2026): e70176, https://doi.org/10.1002/aet2.70176.



Title: Predictors of clinically significant underlying etiologies in children presenting with acquired torticollis

Category: Pediatrics

Keywords: torticollis, neck pain, fever (PubMed Search)

Posted: 9/18/2026 by Jenny Guyther, MD (Updated: 9/18/2026)

This was a retrospective review of children < 18 years who presented to the pediatric emergency department with torticollis in a single center over a 6 year period.

Clinically significant underlying etiologies included infection, tumor, or atlantoaxial rotary fixation requiring surgery (AARF).

352 children with a median age of 4.8 years presented with torticollis and 59 were found to have a significant underlying etiology.

Children with infectious causes were younger and more likely to be febrile and have an elevated CRP.  Infections included retropharyngeal abscess, lymphadenitis and cervical spine osteomyelitis.  AARF was associated with prolonged symptoms, older age, midline tenderness and restricted motion.  Tumor related cases often featured additional red flag symptoms such as headache and vomiting and symptom duration ranging from 5 days to 3 months.

Show References

Shemer M, Noyman S, Rimon A, Cohen N. Predictors of clinically significant underlying etiologies in children presenting with torticollis. Paediatr Child Health. 2026 Jan 12;31(2):111-119. doi: 10.1093/pch/pxaf113. PMID: 41821840; PMCID: PMC12976673.



Title: Arterial complication after hip fracture

Category: Trauma

Posted: 9/17/2026 by Robert Flint, MD (Updated: 9/24/2026)

This case report describes a 2 week delayed hemorrhage from a lacerated profunda femoris artery after an intertrochanteric fracture that requires ORIF. The patient represented with anemia, pain and swelling in the surgically repaired hip. Pulses were still present. The authors conclude: 

“The profunda femoris artery is the vessel most commonly involved in vascular complications associated with proximal femoral fractures because of its close anatomical relationship with the lesser trochanter. Delayed presentations may occur weeks after injury as repeated mechanical irritation from a displaced bone fragment leads to pseudoaneurysm formation and subsequent rupture.”

Show References

https://doi.org/10.1177/14604086261481188



Title: Is EMS documentation of bystander CPR accurate?

Category: EMS

Keywords: 911, dispatch, CPR, bystander (PubMed Search)

Posted: 9/16/2026 by Jenny Guyther, MD (Updated: 9/24/2026)

The national cardiac arrest registry includes information on bystander CPR.  An EMS clinician currently enters bystander CPR (B-CPR) information into the prehospital care report.  The accuracy of this documentation has not been validated.  This study compared EMS documented B-CPR rates to the bystander CPR rates on the 911 audio recordings of the calls in Birmingham Alabama.

Show Additional Information

EMS documentation in the Birmingham CARES registry recorded a B-CPR rate of only 12.3%, while review of corresponding 9-1-1 audio recordings identified a substantially higher rate of 27.5% — more than double the EMS-documented rate.

Of 236 adult non-traumatic out-of-hospital cardiac arrest (OHCA) cases analyzed, 56 cases (23.7%) had disagreements between EMS documentation and 9-1-1 audio review, while 180 cases (76.3%) were concordant. 

In 46 of the 56 discordant cases, 9-1-1 audio confirmed that B-CPR was performed but EMS did not document it. The majority of these involved callers who initiated CPR but discontinued it before EMS arrival, making it invisible to arriving crews. Only 7 of these 46 appeared to be outright EMS misclassifications.

In 10 cases, EMS documented B-CPR but the 9-1-1 audio did not support it — all involved calls that ended before EMS arrival without cardiac arrest recognition or CPR instruction by the dispatcher

Bottom line: Sustained telecommunicator CPR instruction matters through EMS arrival,  as early caller discontinuation of CPR was the primary driver of undercounting, and better dispatcher engagement could both improve CPR continuity and documentation accuracy.

Show References

Coute RA, Smith T, Nathanson BH, Richardson JD, Ferguson WC, Strickland JD, von Schweinitz B, Jackson EA. Discrepancies in Bystander CPR Documentation: Comparing the Birmingham CARES Data with 9-1-1 Audio Review. Prehosp Emerg Care. 2026;30(3):409-413. doi: 10.1080/10903127.2025.2584506. Epub 2025 Dec 9. PMID: 41247107; PMCID: PMC12969134.



Title: SUSPECT Cardiogenic Shock: A Mnemonic to Help Make the Diagnosis

Category: Critical Care

Keywords: cardiogenic shock; cardiology; shock (PubMed Search)

Posted: 9/15/2026 by Shoshana Rudin, MD (Updated: 9/24/2026)

Cardiogenic Shock = a cardiac disorder that results in both clinical and biochemical evidence of sustained tissue hypoperfusion (1).

Cardiogenic shock has high mortality (approx 35-40% in many studies). Mortality worsens at higher stages of cardiogenic shock severity (2,3). Early recognition of cardiogenic shock is critical in order to initiate treatment and transfer patients to an appropriate level of care in a timely manner. 

As an ED physician, you need to SUSPECT cardiogenic shock so that you can ensure appropriate definitive management of your patient (4).

SUSPECT = Symptoms/Signs; Urine Output; Sustained Hypotension; Perfusion; EKG/Echo; Congestion; Triage

Take a look at the Additional Materials to break down this mnemonic in more detail and learn what might make you SUSPECT CS!

Show Additional Information

So what do you need to think about to SUSPECT CS?

Let's break down that mnemonic in a little bit more detail.

Symptoms/Signs:

  • Signs of Shock (Hypoperfusion): AMS, confusion, rapid pulse, cold and clammy extremities, delayed capillary refill
  • Signs of Congestion: Elevated JVP, crackles, rales, lower extremity edema, narrow pulse pressure (<25% of SBP)
  • Symptoms: Chest pain or pressure, chest pain equivalents, orthopnea, paroxysmal nocturnal dyspnea

Urine Output:

  • Oliguria or anuria
    • <0.5 mL/kg/h (roughly <30 mL/h in a 60 kg adult)
  • Place a foley (most accurate hourly UOP), or measure UOP into a urinal, bedpan, Purewick, or condom cath

Sustained Hypotension:

  • SBP <90, MAP <65 for >30 min
  • Or… relative hypotension! >30 mmHg decrease from baseline 
  • Or… anyone who needs pressors or mechanical support to maintain SBP >90
  • Not every patient with cardiogenic shock will have hypotension (yet)!

Perfusion:

  • Lactate: >2 
  • LFTs: ALT >200 U/L or >3x upper limit of normal 
  • Renal function: AKI, AKI on CKD, or Cr >2x upper limit of normal (very concerning)
  • Acidosis: pH <7.2 (very concerning), or metabolic acidosis without another clear etiology
  • You don't need all of the above to be concerned for cardiogenic shock

EKG/Echocardiogram:

  • EKG: STEMI? Arrhythmias? New conduction abnormalities (AV block, LBBB or RBBB)?
  • POCUS: Visually estimate EF, evaluate for LV and/or RV dilation and systolic dysfunction, look for regional wall motion abnormalities
  • More detailed echo: assess for valvular pathology, more accurate quantification of EF (LVOT-VTI, Simpson’s Biplane, etc)

Congestion:

  • Assess for presence or absence of congestion based on physical exam and hemodynamics
    • LV congestion: think about pulmonary edema
    • RV congestion: think about extremity edema, cardiac ascites, elevated JVP, hepatic dysfunction
    • Biventricular congestion: all of the above!

Triage:

  • ICU admission!
    • AMI-CS (Acute MI causing cardiogenic shock): activate cath lab
    • HF-CS (decompensated heart failure, either acute or chronic, causing cardiogenic shock): call heart failure team (if available at your hospital) to discuss advanced therapeutics
    • Choose cardiac ICU if available
  • Activate shock team (if available) to discuss advanced therapeutic options
    • No shock team? Call your ICU, call your on-call cardiologist, and talk through options!
  • Consider transfer to a higher level of care if: 
    • You don’t have access to a cath lab or advanced level of cardiology services
    • You don’t have a cardiac ICU or a MICU with capacity to do advanced hemodynamic monitoring 
    • Your patient might need mechanical circulatory support
    • Your patient might need evaluation for surgical treatment options (CABG, valve repair/replacement, etc)

Proposed Classification of Cardiogenic Shock Levels of Care (originally figure 3, Sinha et al)

Consider the resources that your hospital has available when deciding if/where to transfer a cardiogenic shock patient to a higher level of care.

Show References

1. Waksman R, Pahuja M, van Diepen S, et al. Standardized Definitions for Cardiogenic Shock Research and Mechanical Circulatory Support Devices: Scientific Expert Panel From the Shock Academic Research Consortium (SHARC). Circulation. 2023;148(14):1113-1126. doi:10.1161/CIRCULATIONAHA.123.064527

2. Baran DA, Grines CL, Bailey S, et al. SCAI clinical expert consensus statement on the classification of cardiogenic shock: This document was endorsed by the American College of Cardiology (ACC), the American Heart Association (AHA), the Society of Critical Care Medicine (SCCM), and the Society of Thoracic Surgeons (STS) in April 2019. Catheter Cardiovasc Interv Off J Soc Card Angiogr Interv. 2019;94(1):29-37. doi:10.1002/ccd.28329

3. Naidu SS, Baran DA, Jentzer JC, et al. SCAI SHOCK Stage Classification Expert Consensus Update: A Review and Incorporation of Validation Studies. JACC. 2022;79(9):933-946. doi:10.1016/j.jacc.2022.01.018

4. Sinha SS, Morrow DA, Kapur NK, Kataria R, Roswell RO. 2025 Concise Clinical Guidance: An ACC Expert Consensus Statement on the Evaluation and Management of Cardiogenic Shock. JACC. 2025;85(16):1618-1641. doi:10.1016/j.jacc.2025.02.018



Title: Number One Cause of Death in Pregnancy in the US?

Category: Obstetrics & Gynecology

Posted: 9/14/2026 by Jennifer Wang, MD

Bottom Line: Move over, post-partum hemorrhage, because cardiovascular causes reign supreme! In a new study based on CDC data from 2005-2024, cardiovascular causes were the number one cause of death in pregnancy overall (well, except for COVID…).

Show Additional Information

This study looked at 20 years of data, based on 14000 deaths recorded in CDC's Pregnancy Mortality Surveillance System and found that cardiovascular causes (which include cardiomyopathy, coronary artery disease, valvular disease, and arrhythmias among others) were the number one over cause of death in pregnancy, causing about a quarter of deaths.

Cardiovascular causes led every five year period from 2005 to 2024 except 2020-2024, when infection/sepsis was number one, primarily because of COVID-19, which personally caused almost 60% of pregnancy deaths secondary to infection/sepsis.

Immediately following cardiovascular causes comes infection/sepsis, which causes around 17.8% overall, then other non-cardiovascular medical causes (12%), and then hemorrhage, at only around 11%.

That's not to say that hemorrhage isn't still a big killer - it is, but it's important for emergency providers to keep in mind that pregnancy doesn't do away with all other diseases - a pregnant patient can have as many non-pregnancy problems as they dang well please, whether that's CAD, kidney disease, PEs, or your good, old-fashioned COVID, so always keep looking!

Show References

Njie F, Hollier LM, Gibson C, et al. Trends in Pregnancy-Related Mortality in the United States: Pregnancy Mortality Surveillance System, 2005-2024. Obstet Gynecol. Published online August 7, 2026. doi:10.1097/AOG.0000000000006396



Title: Hydrofluoric acid exposure from Li battery fires

Category: Toxicology

Keywords: hydrofluoric acid exposure (PubMed Search)

Posted: 9/13/2026 by Robert Flint, MD

There is increasing evidence that burning lithium-ion batteries produce hydrofluoric acid (HF) and other fluoridated compounds.  HF is a weak acid and thus it can penetrate deeper into tissue before causing complications. HF exposure causes irritation to the mucosa/skin and leads to pain out of proportion to the appearance of the burn/exposure.  These exposures present a unique risk to first responders and care should be taken to wear appropriate PPE during these events. Regular medical gloves do not protect from exposure. Respiratory protection must be worn to prevent inhalation. Click for treatment options.

Show Additional Information

Treatment is:

-decontamination- remove contaminated clothing and copious irrigation. To prevent secondary exposures, chemical resistant gloves and masks should be worn. Regular medical gloves do not prevent exposure! 

-For skin exposure, calcium gluconate gel applied and massaged into the skin deactivates the HF and corrects the local electrolyte imbalances.

-For pulmonary exposure, nebulized 2.5% calcium gluconate 

-For high level exposures, be aware that there are significant electrolyte shits that can lead to arrythmias and cardiac arrest. Again, calcium is the antidote.

Show References

  1. Cherry Song, Michael Marano, Robin Lee, Christina Lee, Mukosolu Ndubisi, Mostafa Elbahrawy, Ayodeji Folarin, 954 Hydrofluoric Acid Fumes Associated with Electric Vehicle Lithium Ion Battery Fires, Journal of Burn Care & Research, Volume 46, Issue Supplement_1, March/April 2025, Page S365, https://doi.org/10.1093/jbcr/iraf019.485
  2.  Larsson, F., Andersson, P., Blomqvist, P. et al. Toxic fluoride gas emissions from lithium-ion battery fires. Sci Rep 7, 10018 (2017). https://doi.org/10.1038/s41598-017-09784-z
  3.  Stephen Keelan, Maria Murphy, Mark Abrahams, Odhran Shelley, Sharon Kennedy,
    Hydrofluoric acid inhalation injury after electric bike battery fire,
    Journal of Plastic, Reconstructive & Aesthetic Surgery,Volume 118, 2026, Pages 424-427, ISSN 1748-6815, https://doi.org/10.1016/j.bjps.2026.04.035


Title: Serum uric acid in the diagnosis of gout

Category: Orthopedics

Posted: 9/12/2026 by Brian Corwell, MD (Updated: 9/24/2026)

Sensitive or specific enough for diagnosis???

Show Additional Information

Gold standard diagnosis remains crystal analysis by polarized light microscopy.

Why can’t we reply on the serum uric acid level?

Elevated uric acid is common and affects >20% of men and 4% of women. The majority of these individuals never develop gout.

This was seen in a large meta-analysis, of asymptomatic patients with hyperuricemia over 15 years of follow-up.  

Incidence of gout by baseline serum urate: 1.1% (95% CI 0.9–1.4) for <6 mg/dL, rising in a concentration-dependent way to 49% (95% CI 31–67) for ?10 mg/dL. 

As only about half of those with serum urate ?10 mg/dL develop clinically evident gout over 15 years, this implies that prolonged hyperuricemia and additional factors are needed for gout to clinically manifest.

What about during a flare?

A normal value during a flare actually has limited negative predictive value. 

Serum urate frequently falls into the "normal" range during an acute attack, due to the uric acid lowering effect of the acute-phase inflammatory response and increased renal excretion

How to use the serum value:

As one component of clinical diagnosis. 

When crystal analysis is unavailable, serum urate contributes to validated diagnostic algorithms such as the Janssens diagnostic rule. The rule relies heavily on the uric acid value as it receives the single largest weight in the scoring system.

This clinical prediction rule combines male sex, hx of a prior attack, onset within 1 day, joint redness, first MTP involvement, hypertension/cardiovascular disease, and high serum urate.

A score <4 rules out gout in >97% of patients

https://www.mdcalc.com/calc/2175/acute-gout-diagnosis-rule

Take home: Use serum uric acid level to support (not establish) the diagnosis and to guide therapy. BUT, confirm with joint aspiration and crystal analysis whenever feasible, especially to exclude septic arthritis or pseudo gout.

Show References

Dalbeth N, Phipps-Green A, Frampton C, Neogi T, Taylor WJ, Merriman TR. Relationship between serum urate concentration and clinically evident incident gout: an individual participant data analysis. Ann Rheum Dis. 2018 Jul;77(7):1048-1052. doi: 10.1136/annrheumdis-2017-212288. Epub 2018 Feb 20.



Title: Steroid administration, timing, and dose in patients with septic shock in the emergency department

Category: Pharmacology & Therapeutics

Keywords: Emergency department; Hydrocortisone; Sepsis; Septic shock; Steroid; Steroid dose; Steroid timing; Vasopressin interaction (PubMed Search)

Posted: 9/10/2026 by Madison Savidge (Updated: 9/24/2026)

A retrospective analysis of a multicenter prospective cohort study evaluated the association between steroid administration, including hydrocortisone timing and dose, and mortality in patients with septic shock in the ED.

Study findings: Steroid use was associated with lower 28-day mortality (p = 0.001). This association remained in the vasopressin subgroup (p < 0.001) but not in patients who did not receive vasopressin (p = 0.769). A higher norepinephrine-equivalent dose at first steroid administration, a longer interval from first vasopressor initiation to first steroid administration, and hydrocortisone dosing > 300 mg/day were associated with increased mortality.

Bottom line: Steroid administration in the ED was associated with lower 28-day mortality among patients with septic shock, particularly those receiving vasopressin.

Show References

Ahn S, et al. Steroid administration, timing, and dose in patients with septic shock in the emergency department: retrospective analysis of a multicenter prospective cohort study. Sci Rep. 2026 Jun 13;16(1):26929. doi: 10.1038/s41598-026-56521-6.



Title: Renal Resistive Index-Guided MAP titration in Sepsis

Category: Critical Care Literature Update

Keywords: Renal Resistive Index, Sepsis, MAP, Ultrasound (PubMed Search)

Posted: 9/8/2026 by Scott Sparks, MD (Updated: 9/24/2026)

Reducing 28-day all-cause mortality compared to conventional MAP management in sepsis patients requiring vasopressors after initial resuscitation.

  • Cardiac output, MAP, and organ-specific vascular resistance determine organ perfusion.
  • SEPSISPAM trial showed that a higher MAP reduced need for renal replacement therapy.
  • An elevated Renal Resistive-Index (RRI) > 0.7 is associated with impaired perfusion, development of AKI, and increased MR.
  • The kidney is a low vascular resistance organ sensitive to hypoperfusion in sepsis. 
  • 28-day all-cause MR and ventilator free days was decreased. 
  • RRI-guided MAP titration did not specifically decrease MR, incidence of AKI or need for CRRT.
  • Although a pilot trial, this study lends credence to more individualized sepsis hemodynamic management and the need for a larger study.
  • Use of RRI-guided MAP titration in sepsis may decrease overall 28-day MR.
  • How to Guide. https://ultrasoundpaedia.com/renal-arteries-normal/

Show References

Wang C, et al. Renal resistive index-guided mean arterial pressure titration in sepsis: a prospective single-center, single-blind, parallel group randomized controlled trial. Nature Communications. 2026; 17:8759.



Title: Bova Score Refresher

Category: Pulmonary

Posted: 9/6/2026 by Robert Flint, MD

Reminder that the Bova Score (2 points each for heart rate over 110, SBP less than 90, RV strain on CT or echo, and elevated troponin) along with an elevated lactate and BNP can help predict outcomes in hemodynamically stable PE patients. 

"Low risk (score <2): 
Proceed with standard anticoagulation therapy. Standard outpatient or inpatient monitoring, as appropriate.

Intermediate risk (score 3–4):  Consider closer monitoring (e.g., stepdown/ICU). Discuss the possibility of advanced therapies (e.g., thrombolysis) in select cases, especially if clinical status worsens. 

High risk (score >4): Maintain continuous ICU monitoring with a clear plan for rapid intervention if clinical status worsens (e.g., hypotension, worsening hypoxia, arrhythmias). Strongly consider multidisciplinary team involvement and potential advanced therapies (e.g., systemic thrombolysis or catheter-directed therapies)."

Show References

https://www.mdcalc.com/calc/4004/bova-score-pulmonary-embolism-complications



Title: Post termination family needs.

Category: EMS

Posted: 9/5/2026 by Robert Flint, MD (Updated: 9/24/2026)

A structured interview was conducted with families who had a loved one undergo a field terminated cardiac arrest in Denmark. What they found was pre-arrival instructions were very valued. And “After unsuccessful resuscitation, compassionate and unhurried communication was considered crucial.” I am sure this applies to prehospital and in hospital situations alike. Empathy and time are what these families need.

Show References

Skovbo, A., Jørck, C.L., Huniche, L. et al. When prehospital resuscitation attempts are unsuccessful: care for the relatives following termination of treatment in out-of-hospital cardiac arrest. Scand J Trauma Resusc Emerg Med(2026). https://doi.org/10.1186/s13049-026-01689-z



Title: Predicting Agitation in Pediatric Emergency Psychiatric Patients

Category: Pediatrics

Keywords: Agitation, Pediatrics, BRACHA-S, Aggression, Psychiatric emergencies, restraints (PubMed Search)

Posted: 9/4/2026 by Mary Girgis, MD (Updated: 9/4/2026)

BRACHA-S is a 5 item scoring tool that demonstrated moderate discrimination and identified clinically meaningful risk gradients for agitation requiring intervention (ARI) that may support early safety planning.

Show Additional Information

Objectives: To validate the 5 item Brief Rating of Aggression in Children and Adolescents- Short (BRACHA-S) completed by ER nurses for patients presenting with mental and behavioral health encounters for predicting agitation requiring intervention.

Background: MBH concerns account for about 13% of all pediatric ER visits and approximately 10% require the use of physical or pharmacologic restraints.

Methods: They conducted a prospective observational prognostic validation study in 2 pediatric EDs. After patient arrival with MBH concern, ED nurses completed the BRACHA-S. Discrimination was assessed with AUROC and performance was estimated for a 3 tier model. 

Results: Among 472 encounters, 55 (11.7%) had ARI. BRACHA-S scores were associated with ARI (AUROC, 0.72; 95% CI, 0.64–0.79). ARI incidence increased with score: 0, 3.3% (2/60); 1, 6.3% (6/96); 2, 6.4% (7/110); 3, 12.4% (17/137); 4, 31.8% (14/44); and 5, 36.0% (9/25). Scores 0 to 1 had a negative predictive value of 96.7% but a specificity of 13.9%. Scores 4 to 5 had a specificity of at least 89% and a positive predictive value of greater than 33%. Compared with scores 0 to 2, relative risk of ARI was 2.2 (95% CI, 1.1–4.3) for score 3 and 5.9 (95% CI, 3.3–10.7) for scores 4 to 5.

Conclusions: In pediatric ED MBH encounters, the nurse-completed triage BRACHA-S demonstrated moderate discrimination and identified clinically meaningful risk gradients for ARI that may support early safety planning.

Show References

Lynn Babcock, Lily Klein, Yin Zhang, Nancy Daraiseh, Ryan Siders, Beatrice Thomas, Ryan Murphy, Holly R. Hanson, Bijan Ketabchi, Shelby Tanguay, Drew Barzman, Wendy J. Pomerantz; A Brief Tool to Predict Agitation in Pediatric Emergency Psychiatric Patients. Pediatrics August 2026; 158 (2): e2026076729. 10.1542/peds.2026-076729



Title: Get the Lead Out

Category: Toxicology

Keywords: Lead poisoning, Chelation, Neurotoxin (PubMed Search)

Posted: 9/3/2026 by Kathy Prybys, DO

Bottom Line:

Lead has no biologic role in the human body. Lead toxicity and accumulation in humans remains a major health concern. Children < 6 years old are especially vulnerable to lead poisoning.  Lead is a potent neurotoxin and even low levels can cause irreversible harmful neurologic effects in children. Blood lead level of 3.5 µg/dL requires action in children.

Show Additional Information

  • Lead has no biologic role in the human body unlike essential metals (ie. iron, calcium, magnesium, zinc).
  • Children < 6 years old are especially vulnerable to lead poisoning due to environmental and physiological variables
    • Increased hand to mouth activity, 4-5 times better GI absorption, developing nervous systems, bone growth
  • Lead is toxic to every organ system and can present as a multisystem disease with nonspecific clinical manifestations 
  • The nervous system is particularly sensitive to lead and even low levels can cause irreversible harmful neurologic effects in children
    • Decreased IQ, ability to pay attention, and negative behavioral effects
  • Severe acute neurotoxicity is seen at high levels (75-100 ug/dL) causing encephalopathy, seizure, coma
  • No safe Blood Lead Level (BLL) in children has been identified 
  • BLL of 3.5 µg/dL requires action in children
    • Serial monitoring
    • Environmental investigation to identify potential sources of lead
    • Abdominal x-ray and decontamination for BLL > 20-44 ug/dL
  • BLLs do not accurately predict total body burden or duration of exposure but are surrogate markers indicating exposure and absorption 
  • Lead distributes to different body compartments. T1/2 of Lead:
    • Blood = 28 days (99% bound to RBCs)
    • Soft tissue = 40 days
    • Bone =  25 years or greater
  • 90% of total body lead burden is stored in bone which can later  be released 
  • Chelation therapy is indicated for treatment of severe symptoms or markedly elevated BLL:
    • Adults: 100 ug/dL
    • Children: >45-75 ug/dL
  • Acute lead encephalopathy is a medical emergency requiring aggressive chelation therapy

Show References

Recommended Actions Based on Blood Lead Level. August 21, 2025. https://www.cdc.gov/lead-prevention/hcp/clinical-guidance/index.html

The effect of lead exposure on IQ test scores in children under 12 years: a systematic review and meta-analysis of case-control studies. Heidari S, Mostafaei S, Razazian N, et al. Syst Rev. 2022 May 30;11(1):106. doi: 10.1186/s13643-022-01963-y. PMID: 35637522; PMCID: PMC9150353.

Pediatric Lead Chelation Managed During Critical Medication Shortages: Case Report and Literature Review. Singh MK, Kane JM, Said SJ. The Journal of Pediatric Pharmacology and Therapeutics. 2024 Sep;29(5):544–549. doi:10.5863/1551-6776-29.5.544.



Title: What size blade should be used for intubation?

Category: Critical Care

Keywords: airway, laryngoscopy, blade size, critical care (PubMed Search)

Posted: 9/1/2026 by Kristyn McLeod, MD

While many intubations in critically ill patients go smoothly, they are a high risk procedure that EM and ICU physicians seek to continuously improve upon. Blade size is one aspect of the procedure that has received attention over the past several years. We don’t have prospective trials addressing this question, but there are several retrospective analyses that have investigated it.

  • For Direct Laryngoscopy:
    • The 2022 MacSize-ICU study and a 2023 study by Landefeld and colleagues with the Pragmatic Critical Care Research Group suggested that when using Macintosh geometry blades for direct laryngoscopy there was improved first pass success with size 3 blade. Secondary outcomes for these studies point to a worse glottic view with a size 4 blade.
  • For Video Laryngoscopy:
    • This finding did not hold up in video laryngoscopy.  A 2023 study by Park et al. and now a 2026 retrospective study again by Landefeld et al. that looked at blade size for video laryngoscopy with Macintosh geometry blades found no difference in first pass success with size 3 vs size 4 blades.

Bottom Line: Continue to use your best clinical judgment when selecting a blade size, especially when performing video laryngoscopy. When performing direct laryngoscopy, if you are on the fence, consider using size 3 blade.

*These studies do not address intubation with hyperangulated designs

Show References

  • Godet, Thomas, et al. "Impact of Macintosh blade size on endotracheal intubation success in intensive care units: a retrospective multicenter observational MacSize-ICU study." Intensive Care Medicine 48.9 (2022): 1176-1184.
  • Landefeld, Kevin R., et al. "Effect of laryngoscope blade size on first pass success of tracheal intubation in critically ill adults." Critical Care Explorations 5.3 (2023): e0855.
  • Park, Jeongyong, et al. "Effect of blade size on the first-pass success rate of endotracheal intubation using the C-MAC video laryngoscope." Journal of Clinical Medicine 12.22 (2023): 7055.
  • Landefeld, Kevin R., et al. "Blade Size and Outcomes with Standard-Geometry Video Laryngoscopy in Emergency Tracheal Intubations: Secondary Analysis of Two Randomized Controlled Trials." CHEST Critical Care (2026): 100257.


Title: Hyperbaric Oxygen: A beneficial adjunct in burn care

Category: Trauma

Keywords: hyperbaric, HBOT, HBO2, burn, thermal burn (PubMed Search)

Posted: 8/31/2026 by TJ Gregory, MD (Updated: 9/24/2026)

Bottom Line: Burn care is directed by the managing surgeons at regional burn centers. Where available, hyperbaric oxygen treatment can be a beneficial adjunct.

Studies and clinical experience of hyperbaric oxygen treatment in burns has shown modulation of burn shock, dampening of the inflammatory response, reduction of edema, reversal of the zone of stasis, reduction of ischemia and ischemic necrosis, prevention of progression of partial- to full-thickness injury, modulation of inflammation, effecting modulation of hyper-metabolic syndrome, lessening of the capillary leak, preservation of dermal elements, a reduced need for grafting, shortened hospital stay, reduced physiological post-discharge problems, and a reduction in cost of care.

Show References

Cianci P, Sato RM, Faulkner J. Adjunctive Hyperbaric Oxygen in the Treatment of Thermal Burns. Undersea Hyperb Med. 2026 Second Quarter;53(2):363-390. PMID: 42365959.



Title: To admit or not:syncope

Category: Cardiology

Keywords: Syncope (PubMed Search)

Posted: 8/30/2026 by Robert Flint, MD (Updated: 9/24/2026)

This secondary review of a prospective observational group of 1200 syncope/presyncope patients over age 40 found 5.6% experienced a significant adverse outcome within 30 days. These patients had no diagnosis after their ED evaluation. Their conclusion was admission to the hospital “increased the diagnostic yield for SAOs and accelerated time to diagnosis.”

Show References

https://doi.org/10.1111/acem.70393



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