HSF II, S022
Education and Training
09/1992 – 07/1997 Bachelor in Medicine School of Medicine, Fudan University (FormerShanghaiMedicalUniversity), Shanghai, China
09/1997 - 07/2000 Master in Molecular Virology School of Medicine, Fudan University (FormerShanghaiMedicalUniversity), Shanghai, China
09/2002 – 05/2006 Ph.D. in Physiology, Medical College of Georgia, Augusta, GA
Dr. Ying conducted postdoctoral research at the University of Mississippi Medical Center from 2006 to 2007 and then at the Ohio State University Dorothy M. Davis Heart and Lung Research Institute, where he served as an American Heart Association postdoctoral fellow from 2011 to 2013. Dr. Ying joined the faculty at the University of Maryland, Baltimore, in October of 2013.
Air pollution, Cardiovascular diseases, Diabetes, Imflammation
Ying Z,* Allen K, Zhong J, Chen M, Williams KM, Wagner JG, Lewandowski R, Sun Q, Rajagopalan S, Harkema JR. Subacute inhalation exposure to ozone induces systemic inflammation but not insulin resistance in a diabetic mouse model. Inhal Toxicol. 2016;28(4):155-63.
Ying Z*, Xie X, Bai Y, Chen M, Wang X, Zhang X, Morishita M, Sun Q, Rajagopalan S. Exposure to concentrated ambient particulate matter induces reversible increase of heart weight in spontaneously hypertensive rats. Part Fibre Toxicol. 2015 Jun 25;12(1):15.
Ying Z*, Xu X, Bai Y, Zhong J, Chen M, Liang Y, Zhao J, Liu D, Morishita M, Sun Q, Spino C, Brook RD, Harkema JR, Rajagopalan S. Long-term concentrated ambient PM2.5 exposure increases mouse blood pressure through abnormal activation of the sympathetic nervous system: a role for hypothalamic inflammation. Environ Health Perspect. 2014 Jan;122(1):79-86.
Dr. Ying has over 12 years of research experience in cardiometabolic diseases including hypertension, type 2 diabetes, and atherosclerosis. Currently, his research is focused on the cardiometabolic effects of exposure to ambient particulate matter, which is funded by the National Institutes of Health (NIH). In the study, multimodal approaches are used to delineate the mechanism whereby exposure to ambient particulate matter induces abnormalities in blood pressure and insulin sensitivity.